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3.3 Neuroinflammation Neuroinflammation has emerged as a central and biologically plausible contributor to the pathophysiology of depressive disorders, bridging immune dysregulation with alterations in neurotransmission, neuroplasticity, and stress responsivity (30, 31)

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The temporal sequence of changes of hyperglycemia and incretin effect is still debated with conflicting results (6, 7)

While the cases are consolidated before a single federal judge for pretrial proceedings, like discovery and key legal rulings, each plaintiffs case ultimately stands on its own